T Mitochondriaonthemove:BMSCsfuelAMLenergy

S pecific cancer cells , notably grade II/III glioma (35, 36), secondary glioblastoma (127), and acute myeloid leukemia (AML) (16, 60, 103, 144) cells, exhibit heterozygous

Speci fi city of PCR-based clonality analysis of immunoglobulin heavy chain gene rearrangements for the detection of bone marrow involvement by low-grade B-cell lymphomas. Evaluation

Human bone marrow multipotent mesenchymal stromal cells (hBM-MSC) represent an appealing source of adult stem cells for cell therapy and tissue engineering.. Since hBM-MSC are

AML = acute myelogenous leukemia; BC = breast cancer; CRC = colorectal cancer; GIST = gastrointestinal stromal tumor; HCC = hepatocellular carcinoma; MBC = metastatic breast

The versatility of cAMP signaling is highly represented in hematopoietic cells, with induction of apoptosis in thymocytes [222, 223] and the myeloid leukemia (AML) cell

The aim of this study was to evaluate bone regeneration using bone marrow-derived mesenchymal stromal cells (MSCs) in a clinical trial, a less invasive approach than autologous

Phase I/II study of volasertib (BI 6727), an intravenous polo-like kinase (Plk) inhibitor, in patients with acute myeloid leukemia (AML): updated results of the dose fi nding phase

The aim of the present study was to determine whether direct communication between bone marrow stromal cells (MSC) and human umbilical vein endothelial cells (EC) could influence