Mesenchymal stem cells support survival and Proliferation of Primary human acute Myeloid leukemia cells through heterogeneous Molecular Mechanisms

The aim of this study was to assess the viability and osteogenic differentiation of primary human osteoblasts and adipose tissue–derived mesenchymal stem cells from various donors

Conclusion: In the present in vitro study, the isolated dental pulp cells expressed cell surface markers comparable to the expression from bone marrow mesenchymal stem cells

S pecific cancer cells , notably grade II/III glioma (35, 36), secondary glioblastoma (127), and acute myeloid leukemia (AML) (16, 60, 103, 144) cells, exhibit heterozygous

In this issue of Blood, Marlein et al 1 identify a tumor-speci fi c NOX2-dependent transfer of mitochondria from bone marrow stromal cells (BMSCs) to acute myeloid leukemia (AML)

following relapse post autologous stem cell transplantation in adult patients with acute 281. myeloid leukemia: A retrospective analysis of 537 patients from the Acute Leukemia

Release of angiopoietin-1 by primary human acute myelogenous leukemia cells is associated with mutations of nucleophosmin, increased by bone marrow stromal cells and possibly

Supra-additive cytokine levels can be seen in MSC-AML cell cocultures MSC24429, MSC24539 and MSC25200 were cocultured in transwell cultures with primary human AML cells derived from

The aims of this study were twofold: first to determine the in vitro angiogenic and osteogenic gene- expression profiles of endothelial cells (ECs) and mesenchymal stem cells